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Toll-like receptor 2 is normally localized to the cell membrane (green outlines, left panel). However, a KSHV protein affects the normal distribution (diffuse green, right panel). Endoplasmic reticulum is shown in red.
Pathogens entering our body only remain unnoticed for a short period. Within minutes our immune cells detect the invader and trigger an immune response. However, some viruses have developed strategies to avoid detection and elimination by our immune system. Researchers from the Helmholtz Centre for Infection Research (HZI) in Braunschweig have now been able to show how the herpesviruses achieve this.
The Kaposi’s sarcoma-associated herpesvirus (KSHV), a gammaherpesvirus that can cause multiple forms of cancer, establishes lifelong infections within the body. To do so the virus has to find a way to modulate the immune system of its host.
“Intruders are usually fought off immediately by an antiviral immune response that is triggered by sensors including the toll-like…
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